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Researchers from the Harvard Extension School and Pacific Northwest University examined the substantial body of evidence connecting Vitamin K2 factors involved in Alzheimer’s Disease (AD) pathogenesis.
After considering the antiapoptotic and antioxidant effects of Vitamin K2 and its impact on neuroinflammation, mitochondrial dysfunction, cognition, cardiovascular health, and comorbidities in AD, the researchers concluded that this demonstrates K2 as having the potential to slow AD progression and “contribute to its prevention.”
This is the first review to consider the physiological roles of Vitamin K2 in the context of AD, and the authors write, “given the recent shift in Alzheimer’s Disease research toward nonpharmacological interventions, our findings emphasise the timeliness and need for clinical studies involving vitamin K2.”
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